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Processes | Free Full-Text | Competing Endogenous RNAs in Cervical Carcinogenesis: A New Layer of Complexity | HTML
Modification of miRNA Expression through plant extracts and compounds against breast cancer: Mechanism and translational significance - ScienceDirect
Glucose and oleic acid mediate cellular alterations in GLP‐1‐induced insulin‐positive differentiating UCBMSCs | Request PDF
PDF) Deciphering the Molecular Role of Non-coding RNA in Malignant Cardiac Disease | published in Biointerface Research in Applied Chemistry
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PDF) Exposomes to Exosomes: Exosomes as Tools to Study Epigenetic Adaptive Mechanisms in High-Altitude Humans
LncRNA NEAT1 knockdown ameliorates LPS-induced human kidney injury by mediating the miR-330-5p/FOXO3 axis | SpringerLink
LncRNA NEAT1 knockdown ameliorates LPS-induced human kidney injury by mediating the miR-330-5p/FOXO3 axis | SpringerLink
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LncRNA NEAT1 knockdown ameliorates LPS-induced human kidney injury by mediating the miR-330-5p/FOXO3 axis | SpringerLink
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Processes | Free Full-Text | Competing Endogenous RNAs in Cervical Carcinogenesis: A New Layer of Complexity | HTML
Processes | Free Full-Text | Competing Endogenous RNAs in Cervical Carcinogenesis: A New Layer of Complexity | HTML
VORTEX | Racing Kart - leader store in the kart world
Lockdown of mitochondrial Ca2+ extrusion and subsequent resveratrol treatment kill HeLa cells by Ca2+ overload - ScienceDirect
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Lockdown of mitochondrial Ca2+ extrusion and subsequent resveratrol treatment kill HeLa cells by Ca2+ overload - ScienceDirect
Lockdown of mitochondrial Ca2+ extrusion and subsequent resveratrol treatment kill HeLa cells by Ca2+ overload - ScienceDirect
IJERPH | Free Full-Text | Exposomes to Exosomes: Exosomes as Tools to Study Epigenetic Adaptive Mechanisms in High-Altitude Humans | HTML
IJERPH | Free Full-Text | Exposomes to Exosomes: Exosomes as Tools to Study Epigenetic Adaptive Mechanisms in High-Altitude Humans | HTML
LncRNA NEAT1 knockdown ameliorates LPS-induced human kidney injury by mediating the miR-330-5p/FOXO3 axis | SpringerLink
Frontiers | Endoplasmic Reticulum-Mitochondria Contacts: A Potential Therapy Target for Cardiovascular Remodeling-Associated Diseases
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